Quick Comparison

B-ComplexLion's Mane
Half-LifeWater-soluble; excreted daily (except B12 which is stored)Bioactive compounds (hericenones, erinacines) accumulate with daily use; effects are cumulative
Typical DosageStandard: A quality B-complex providing 25-100 mg of B1, B2, B3, B5, B6, plus 400-800 mcg folate (as methylfolate) and 500-1000 mcg B12 (as methylcobalamin). Methylated forms preferred for B9 and B12 (folate → methylfolate, B12 → methylcobalamin). Take in the morning — B vitamins can be mildly energizing.Standard: 500-3000 mg daily of fruiting body extract. For NGF stimulation: look for extracts containing both hericenones (from fruiting body) and erinacines (from mycelium). Dual-extract products provide both. Take consistently for 4+ weeks for noticeable effects.
AdministrationOral (capsules, tablets, sublingual). Methylated forms preferred for B9 and B12. Take with breakfast.Oral (capsules, powder, tincture, whole mushroom). Extracts standardized for beta-glucans and/or hericenones are preferred.
Research Papers10 papers9 papers
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Mechanism of Action

B-Complex

Each B vitamin serves specific neurological functions: B1 (thiamine) — cofactor for transketolase (pentose phosphate pathway), pyruvate dehydrogenase, and alpha-ketoglutarate dehydrogenase; essential for glucose metabolism and ATP production in neurons. B2 (riboflavin) — precursor to FAD/FMN, cofactors for Complex I and II of the electron transport chain, and glutathione reductase. B3 (niacin/niacinamide) — precursor to NAD+/NADPH via the salvage pathway; NAD+ is substrate for sirtuins, PARP, and 400+ dehydrogenases. B5 (pantothenic acid) — component of coenzyme A, required for acetylcholine synthesis via choline acetyltransferase and for fatty acid oxidation. B6 (pyridoxine) — cofactor for AADC (5-HTP to serotonin, L-DOPA to dopamine), GABA synthesis (GAD), and homocysteine metabolism. B9 (folate) — tetrahydrofolate donates methyl groups for dTMP and purine synthesis, and for homocysteine remethylation. B12 (cobalamin) — cofactor for methionine synthase (myelin maintenance) and methylmalonyl-CoA mutase.

Lion's Mane

Lion's Mane contains two classes of bioactive compounds: hericenones (A-H, found in the fruiting body) and erinacines (A-I, found in the mycelium). Both stimulate the synthesis of nerve growth factor (NGF) in astrocytes and neurons — hericenones may act through enhancement of NGF gene expression, while erinacines cross the blood-brain barrier and directly induce NGF. NGF binds to TrkA receptors and is essential for the survival, maintenance, and regeneration of cholinergic neurons, particularly in the hippocampus and basal forebrain. This promotes neurogenesis, dendritic arborization, and remyelination of nerve fibers. Lion's Mane also reduces neuroinflammation through inhibition of NF-κB signaling and suppression of pro-inflammatory cytokine production. It may enhance BDNF expression and support the gut-brain axis.

Risks & Safety

B-Complex

Common

Bright yellow urine (harmless — riboflavin excretion), mild nausea.

Serious

Very safe at standard doses. B6 can cause peripheral neuropathy at >200 mg daily for extended periods.

Rare

Flushing from niacin (B3) if non-flush form is not used.

Lion's Mane

Common

Mild gastrointestinal discomfort, itching (possibly from NGF stimulation).

Serious

Allergic reactions in people with mushroom allergies.

Rare

Exacerbation of asthma symptoms, skin rash.

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